Hyperparathyroidism, osteomalacia, adynamic bone disease, and β2 microglobulin amyloidosis are all conditions involving bone that may be observed in chronic renal failure patients. Secondary hyperparathyroidism is still the most frequent cause of clinically expressed renal bone disease, despite the improvement achieved in its treatment and prevention (Cannata, 1998). Disturbance of the metabolism of calcium, phosphorus and vitamin D in patients with chronic renal failure plays a key role in the development of secondary hyperparathyroidism (Naveh et al., 1995). The choice of appropriate dialysate Ca concentration is crucial in the management of dialysis patients as an excessive Ca load has been associated with vascular calcifications, whereas Ca depletion can worsen secondary hyperparathyroidism and decrease bone mass. In hemodialysis, Ca transfer by diffusion depends on the concentration gradient between dialysate and blood (Binswanger et al., 1990).