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386907

Exploring the Role of PADI4 rs874881 and rs1635564 in Susceptibility to Systemic Lupus Erythematosus and Lupus Nephritis in Egyptian patients

Article

Last updated: 28 Dec 2024

Subjects

-

Tags

Genetics

Abstract

Background: Systemic lupus erythematosus (SLE) is an autoimmune disease with pathogenesis involving immune complex deposition in various organs resulting in inflammation and tissue damage. Lupus nephritis (LN) is a crucial clinical manifestation of SLE. Immune complex deposition leads to neutrophil activation and death with neutrophil extracellular traps (NETs) generation. NETs are formed of histones, chromatin fibrils and antimicrobial peptides. Chronic NETs formation (NETosis) aggravates inflammatory tissue and is believed to be dependent on histone citrullination. Peptidylarginine deiminase-4 (PADI4) enzyme is essential for citrullination and highly expressed by neutrophils. PADI4 functionality and expression might be influenced by single-nucleotide polymorphisms (SNPs) in PADI4 gene. Objective: The aim of this work is to study the possible association of PADI4 rs874881 and rs1635564 SNPs with SLE and LN susceptibility in the Egyptian population. Methodology: this case control study encompasses 102 SLE patients (including 52 LN patients) and 50 healthy controls, all were subjected to genotyping analysis of PADI4 rs874881 and rs1635564 SNPs by Real-Time Polymerase Chain Reaction (PCR) technique. Results: Concerning PADI4 rs874881 polymorphism, genotype and allele frequencies did not show any significant differences between SLE patients and healthy controls (p=0.1 and 0.2 respectively), or between SLE patients with or without LN (p= 0.1 and 0.9 respectively). Regarding PADI4 rs1635564 there was no statistical significance between SLE patients and healthy controls in distribution of genotypes or alleles (p=0.07 and 0.8 respectively), and the same for SLE patients with or without LN with p=0.2 for both of them. Conclusion: PADI4 gene polymorphisms rs874881 and rs1635564 did not show any association with SLE or LN in our sample of Egyptian patients from Cairo.

DOI

10.21608/ejmm.2024.326662.1353

Keywords

Systemic lupus erythematosus, Lupus nephritis, PADI4 polymorphisms, rs874881, rs1635564

Authors

First Name

Ingy

Last Name

Ashmawy

MiddleName

-

Affiliation

Department of Clinical and Chemical Pathology, Medical Research and Clinical Studies Institute, National Research Centre, Cairo, Egypt

Email

ingyashmawy@ymail.com

City

Giza

Orcid

0000-0002-6598-3568

First Name

Abeer

Last Name

Ramadan

MiddleName

-

Affiliation

Department of Molecular Genetics and Enzymology, Human genetics and genome research institute, National Research Centre, Cairo, Egypt

Email

dr_aramadan1978@yahoo.com

City

Cairo, Egypt

Orcid

-

First Name

Maha

Last Name

Abdelhadi

MiddleName

-

Affiliation

Internal Medicine Department, Medical Research and Clinical Studies Institute, National Research Centre, Cairo, Egypt

Email

mahahadi39@yahoo.com

City

Cairo, Egypt

Orcid

-

Volume

34

Article Issue

1

Related Issue

50252

Issue Date

2025-01-01

Receive Date

2024-10-07

Publish Date

2025-01-01

Print ISSN

1110-2179

Online ISSN

2537-0979

Link

https://ejmm.journals.ekb.eg/article_386907.html

Detail API

https://ejmm.journals.ekb.eg/service?article_code=386907

Order

386,907

Type

New and original researches in the field of Microbiology.

Type Code

2,038

Publication Type

Journal

Publication Title

Egyptian Journal of Medical Microbiology

Publication Link

https://ejmm.journals.ekb.eg/

MainTitle

Exploring the Role of PADI4 rs874881 and rs1635564 in Susceptibility to Systemic Lupus Erythematosus and Lupus Nephritis in Egyptian patients

Details

Type

Article

Created At

28 Dec 2024