Background: Intracerebral hemorrhage (ICH) remains the second leading cause of stroke accompanied with great mortality rate and severe neurological deficits. For ICH patients, available treatment options are still limited, therefore early recognition and improving results requires careful management of potential sources of harm. Objective: Review of literature aboutglycemic state and intracerebral hemorrhage. Methods: We searched PubMed, Google Scholar, and Science Direct for relevant articles on glycemic state and intracerebral hemorrhage. However, only the most recent or thorough studies were taken into account between October 2000 and April 2023. The authors also evaluated the value of resources culled from other works in the same genre. Documents written in languages other than English have been ignored due to a lack of translation funds. Unpublished works, oral presentations, conference abstracts, and dissertations were generally agreed upon not to be qualified as scientific research. Conclusion: Patients with diabetes mellitus (DM) appear to develop a self-protective mechanism in response to chronic hyperglycemia by preferentially down-regulating glucose transporters (GLUT-3 as well as GLUT-1), which allows glucose to enter cells independently of insulin and thus reduces the acute fluctuation of glucose concentration and endothelial cell apoptosis. Hyperglycemia due to stress is a temporary condition characterized by higher-than-normal blood sugar levels. Myocardial infarction, ischemic stroke, and intracerebral haemorrhage are all serious illnesses in which this phenomenon is frequently observed.