Background: Hair follicle and sebaceous gland inflammation causes acne, which is a long-term, chronic skin condition. The androgen-induced sebum overproduction leads to first acne appearance. It is caused by altered and aberrant keratinization, inflammation and the colonization of facial hair follicles by the bacteria Propionibacterium acnes. Acne also has a delayed immune response, as well as influences from the environment and genetics. Treatments for post-acne scarring have included topical preparations such as chemical peels, dermabrasion, laser resurfacing and punch excision and punch elevation, as well as fat transfer, implantation of autologous collagen and cultured and expanded autologous fibroblasts. Other treatments have included trichloroacetic acid focal treatment and skin microneedling to improve the dermal extracellular matrix (ECM) proteins. Objective: To determine the updated treatment modalities in atrophic post-acne scars. Conclusion:  In most types of atrophic acne scars combination therapies typically provide superior outcomes than solo treatments.