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236846

The Ameliorative Potential of Alda-1 on Experimentally Induced Liver Fibrosis in Adult Male Mice. A Histological, Immunohistochemical and Biochemical Study

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Last updated: 01 Jan 2025

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Abstract

Introduction: Because of its complex pathogenesis, liver fibrosis remains one of the diseases with no standard treatment. Alda-1 is considered as a promising drug in ameliorating such fibrosis.
Aim of the Work: To evaluate the potential effect of Alda-1 after thioacetamide (TAA)-induced liver fibrosis in male mice.
Materials and Methods: Twenty-five adult male mice (25–27 gm aged 2-3 months) were allocated into five equal groups. Group I (control group), group II (Alda-1 group): each mouse was given Alda-1 [5 mg/kg, intraperitoneally (ip)] twice weekly for four weeks. Group III (TAA group): each animal received TAA (200 mg/kg, ip) twice weekly for seven weeks. Group IV (TAA +Alda-1 group): each mouse was given TAA as group III. After stoppage of TAA administration, Alda-1 was given at a dose as group II and continued for four weeks. Group V (recovery group) each animal received TAA treatment as group III and left without any treatment for an extra four weeks. The histological changes were identified by the light (H&E and Masson's trichrome stains) and electron microscopies, immunohistochemical and morphometric analysis. Blood samples were taken to evaluate the liver's function.
Results: TAA caused marked histological and biochemical attenuation of hepatocytes structure and function with significant increase in collagen deposition. In addition, TAA caused significant elevation of liver enzymes. In Alda-1treated In Alda-1treated group (IV), hepatocytes revealed nearly normal structure, significantly decreased the elevated liver enzymes and significant increase in reduced glutathione and decrease in malondialdehyde in liver homogenate. Alda-1 decreased the elevated transforming growth factor, collagen-1 gene expression and the area percentage of collagen. In addition, alpha smooth muscle actin was significantly reduced. The anti-inflammatory effects were also detected by the decrease in the interleukin-6 and tumor necrosis factor-α.
Conclusion: Alda-1 ameliorated TAA-induced liver fibrosis in mice. This might be due to its antioxidant, antifibrotic and anti-inflammatory effects.

DOI

10.21608/ejh.2022.133870.1674

Keywords

Alda-1, fibrosis, hepatocytes, histology, Immunohistochemistry

Authors

First Name

amany

Last Name

solaiman

MiddleName

-

Affiliation

histology and cell biology ,alexandria egypt

Email

amanysolaiman@gmail.com

City

-

Orcid

0000-0002-9406-6152

First Name

silvia

Last Name

Sawires

MiddleName

Kamil Seddik

Affiliation

histology and cell biology, alexandria university, Egypt

Email

silviakamil76@yahoo.com

City

-

Orcid

-

Volume

45

Article Issue

3

Related Issue

37238

Issue Date

2022-09-01

Receive Date

2022-04-19

Publish Date

2022-09-01

Page Start

949

Page End

968

Print ISSN

1110-0559

Online ISSN

2090-2417

Link

https://ejh.journals.ekb.eg/article_236846.html

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https://ejh.journals.ekb.eg/service?article_code=236846

Order

21

Type

Original Article

Type Code

119

Publication Type

Journal

Publication Title

Egyptian Journal of Histology

Publication Link

https://ejh.journals.ekb.eg/

MainTitle

The Ameliorative Potential of Alda-1 on Experimentally Induced Liver Fibrosis in Adult Male Mice. A Histological, Immunohistochemical and Biochemical Study

Details

Type

Article

Created At

22 Jan 2023