A typical scenario of pathogenesis is a lunate with several of the intrinsicanatomical features noted above, predisposing the lunate to elasticdeformation of trabeculae. Microintratrabecular fractures occur and heal,but have certain changes along the plane of injury. With repeated injuriesalong the same plane and resultant rupture of capillary systems, theredevelops a physiological fault plate with increased resistance to normalblood flow . Additional fault plates may form in other areas of the lunate.This phenomenon is probably present in most of our carpal bones, buttime and the avoidance of similar overload injuries preclude developmentof multiple plates.